Left Ventricular Noncompaction Cardiomyopathy as a Potential Cause of Bilateral Posterior Cerebral Artery Stroke – a Rare and Unique Clinical Occurrence

Marwa Elnazeir; Lisa Nobel; Siddharth Narayanan; Fahed Darmoch; Francesco Massari; Timothy Fitzgibbons; Raphael A. Carandang; Adalia Jun-O'Connell

doi:10.12659/AJCR.931103

22 June 2021 : Case report USA

Left Ventricular Noncompaction Cardiomyopathy as a Potential Cause of Bilateral Posterior Cerebral Artery Stroke – a Rare and Unique Clinical Occurrence

Rare coexistence of disease or pathology

Marwa Elnazeir^1ABCDEF*, Lisa Nobel^1ABD, Siddharth Narayanan^2BEF, Fahed Darmoch^3BD, Francesco Massari^4ACDE, Timothy Fitzgibbons^3ABCDE, Raphael A. Carandang^5ACDEF, Adalia Jun-O'Connell^1ABCDEFG

DOI: 10.12659/AJCR.931103

Am J Case Rep 2021; 22:e931103

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Figure 2. Cerebral angiogram post-pharmacological thrombectomy. Superselective microcatheter intra-arterial administration of tissue plasminogen activator resulted in reperfusion of the bilateral posterior cerebral artery (PCA) occlusions, with significant improvement of the antegrade flow within the corresponding PCA vascular territories.

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Left Ventricular Noncompaction Cardiomyopathy as a Potential Cause of Bilateral Posterior Cerebral Artery Stroke – a Rare and Unique Clinical Occurrence

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